07-20-2008, 09:10 PM
white infarct i mean******
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336 mile marathon - pepsi00
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07-20-2008, 09:11 PM
A 25-year-old male patient complains of increased thirst and increased frequency of urination. He denies any trauma or recent surgery. At baseline his urine specific gravity is 1.001 and his serum osmolality is 350.
He is admitted to the hospital with for observation and water restriction. After 6 hours, his urine specific gravity is 1.003 and his serum osmolality is 365. He is given intranasal desmopressin (DDAVP) and water is restricted for 6 hours. Afterward his urine specific gravity is 1.004 and his serum osmolality is 400. What medication most likely caused these symptoms? A. Aspririn B. Furosemide C. Gentamicin D. Lithium E. Penicillin
07-20-2008, 09:14 PM
DDD
By the way hear is PALE infarct. when u see a tree u forget that u are in forest.kidding
07-20-2008, 09:17 PM
D is the correct answer
This patient is suffering from nephrogenic diabetes insipidus which is a side effect of lithium (option D). Diabetes insipidus is characterized by increased thirst (polydipsia) and increased urination (polyuria) with the inability to to concentrate urine due to a lack of ADH. Therefore, urine specific gravity will be low (250. Nephrogenic diabetes insipidus (lack of response to ADH) can be distinguished from central (loss of ADH production) because nephrogenic DI will not respond to DDAVP which is a ADH analog. Furosemide (option B) is a loop diuretic which would cause decreased urine specific gravity but the serum osmolality would not increase as much. Aspirin (option A), Gentamicin (option C) and Penicillin (option E) can cause acute tubular necrosis (ATN) and acute interstitial nephritis which would cause an increase in serum creatinine but would not cause an low urine specific gravity or a high serum osmolality.
07-20-2008, 09:20 PM
---central DI-jus give DDVAP
---nephrogenic give thiazides lithium induceed DI-amioloride
07-20-2008, 09:23 PM
Well Prions-the brain protein is misconfigured in Mad Cow--become sponge
How would our body kill it? CD-8 cells or NK cells But how does our immuno even kill it, if our brain is being attack the commander center,if u take that away,its all down the road from there............................ but what is ur answer |
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